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【公司名稱】 廣州健侖生物科技有限公司
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乍看之下，Davies似乎不像“癌癥戰(zhàn)爭”中的戰(zhàn)士，他是物理學(xué)出身，而非生物醫(yī)學(xué)。但是，大約7年前，他被邀請在亞利桑那州建立一個新機(jī)構(gòu)——由國立癌癥研究所資助的12所機(jī)構(gòu)中的1個，以便將物理學(xué)家和腫瘤學(xué)家聯(lián)合在一起，發(fā)現(xiàn)該疾病的新視角。“我們被要求從上到下重新思考癌癥。”Davies說。
隨后，Davies與澳大利亞國立大學(xué)天體生物學(xué)家Charley Lineweaver和英國倫敦健康科學(xué)中心腫瘤學(xué)家Mark Vincent展開合作，提出了“返祖現(xiàn)象”模式，將癌癥定位為古老“預(yù)編”特性的重新表達(dá)。在上個月發(fā)表于《生物學(xué)論文集》的新研究中，該研究小組指出，因?yàn)榘┌Y出現(xiàn)在許多動物、植物和人類中，那么它必須從億萬年前開始進(jìn)化，那時生物擁有共同的單細(xì)胞祖先。
在那時，細(xì)胞受益于永生，或無限增殖能力，正如癌癥那樣。但當(dāng)復(fù)雜的多細(xì)胞生物開始出現(xiàn)，“‘永生’被轉(zhuǎn)包給卵子和抗原抗體。”Davies說，不涉及繁衍的體細(xì)胞不再需要這種機(jī)能。
該研究小組提出的假設(shè)是，當(dāng)健康細(xì)胞面臨環(huán)境威脅時，例如輻射或生活因素，細(xì)胞能夠回復(fù)到“預(yù)編的安全模式”。這樣一來，細(xì)胞會拋棄更高的機(jī)能，并將它們的休眠能力切換至增殖能力，以便存活下來。“癌癥是一種自動防障功能，”Davies提到，“一旦這個子程序被觸發(fā)，就會冷酷地運(yùn)行該程序。”
9月11日，在英國帝國理工學(xué)院舉辦的一個醫(yī)學(xué)工程會議上，Davies描述了一系列基于這種返祖現(xiàn)象的癌癥療法。Davies指出，與簡單地攻擊癌癥復(fù)制能力不同，該模型揭示了“癌癥的阿喀琉斯之踵”。例如，如果該理論正確，那么癌癥進(jìn)化初期地球的環(huán)境更酸且氧氣更稀薄。因此，研究人員預(yù)測，利用高水平氧氣和在飲食中加入還原糖以降低酸性，能夠抑制腫瘤并引起腫瘤收縮。

At first glance, Davies does not seem like a warrior in the "cancer war", he is a physics background, rather than biomedical. However, about seven years ago he was invited to establish a new agency in Arizona, one of 12 institutions funded by the National Cancer Institute, to bring together physicists and oncologists to discover the disease New perspective. "We were asked to rethink the cancer from top to bottom," Davies said.
Davies then collaborated with astrobiologist Charley Lineweaver of the Australian National University and Mark Vincent, an oncologist at the London Health Science Center in the UK, to propose a "return to ancestor phenomenon" model that positions cancer as a re-expression of the ancient "pre-programmed" nature. In a new study published in the Proceedings of Biology last month, the team pointed out that because cancer appears in many animals, plants and humans, it must evolve hundreds of millions of years ago when the creatures have common Single cell ancestor.
At that time, cells benefit from eternal life, or infinite proliferative capacity, just as cancer does. But when complex multicellular organisms begin to emerge, "Immortality" is subcontracted to the egg and antigenic antibodies, Davies said, not involving multiplying somatic cells that are no longer needed for this function.
The team's hypothesis is that when healthy cells are exposed to environmental threats, such as radiation or life, cells can revert to a "pre-programmed security model." As a result, cells discard higher functions and switch their dormancy to proliferative capacity in order to survive. "Cancer is an automatic barrier," Davies said. "Once the subroutine is triggered, it runs coldly."
On September 11, at a medical engineering conference organized by Imperial College London, Davies described a series of cancer therapies based on this anteversion phenomenon. Davies points out that unlike a simple attack on cancer replication, the model reveals "the Achilles heel of cancer." For example, if the theory is correct, the Earth's environment is more acidic and oxygenier at the beginning of cancer's evolution. Therefore, the researchers predicted that the use of high levels of oxygen and added to the diet reducing sugar to reduce acidity, can inhibit the tumor and cause tumor shrinkage.